1 grand et fort volume in-8 (24.5 x 16.5 cm), (4), 1311 pp., quelques figures dans le texte, demi-chagrin brun de l'époque, dos à nerfs orné de filets dorés, tête dorée, quelques traces de frottement, intérieur en très bon état. Volume devenu rare de cette prestigieuse revue, comportant de nombreuses publications des élèves de Charcot (Babinski, Dejerine, Brissaud, Pierre Marie, Raymond, etc.). /// A RARE NEUROLOGICAL VOLUME CONTAINING FIRST DESCRIPTIONS OF BABINSKI-NAGEOTTE SYNDROME, OF DIDE-BOTCAZO SYNDROME, etc. : 1°). BABINSKI (J.) & NAGEOTTE (J.). Hémiasynergie, latéropulsion et myosis bulbaires avec hémianesthésie et hémiplégie croisées (pp. 358-365, 2 fig.) Cf. Garrison & Morton, 4589 : "Babinski-Nageotte syndrome"; Jablonski S: Eponymic syndromes and diseases, p. 17; Philippon J & Poirier J: Joseph Babinski, a biography, p. 204; Dai S & Pérennou D: Renaissance of "lateropulsion", Annals of Physical and Rehabilitation Medicine, 2021,64(6):101595: "The term lateropulsion was introduced in the medical literature in the early 1900s, by Joseph Babinski (1857–1932) and Jean Nageotte (1866–1948)."; Larner AJ: A Dictionary of Neurological Signs, p. 209. 2°). DIDE (M.) & BOTCAZO. Amnésie continue, cécité verbale pure, perte du sens topographique, ramollissement double du lobe lingual (pp. 676-680, 5 fig.). Cf. Chabrol H & Schmitt L: Maurice Dide, 1873–1944, Am J Psychiatry. 2001 May;158(5):703: "In 1902, he described a neurological syndrome caused by a bilateral obstruction of the posterior cerebral arteries, which is internationally known as Dide-Botcazo syndrome."; Jablonski S: Eponymic syndromes and diseases, p. 81; Lazzarino De Lorenzo LG et al.: The Dide-Botcazo syndrome: forgotten and misunderstood, Cortex. 2014 Jul;56:182-90: "Bilateral infarcts of the posterior cerebral arteries are associated with a range of visual and memory deficits. In 1902, Dide and Botcazo presented a clinico-pathological case study linking visual field defects, topographical disorientation, retro-anterograde amnesia and alexia with bilateral medial occipito-temporal lesions. Based on the findings they suggested the occipital lobe and inferior longitudinal fasciculus played an important role in memory." 3°). DEJERINE M. et Mme (Jules et Augusta). Sur l'hypertrophie compensatrice du faisceau pyramidal du côté sain, dans un cas d'hémiplégie cérébrale infantile (pp. 642-646, 5 fig.) Cette publication novatrice sur les suppléances (à la fois du point de vue fonctionnel et anatomique est basée sur une observation anatomo-clinique. Il s'agit du premier cas d'hypertrophie compensatrice du faisceau pyramidal observée chez l'homme. Cf. Thomas B et al.: Quantitative diffusion tensor imaging in cerebral palsy due to periventricular white matter injury, Brain, 2005,128(11):2562–2577: "Several authors have noted compensatory hypertrophy of the contralateral pyramidal tract (CST) in autopsy studies in adults who had possible congenital hemiparesis (Déjérine and Déjérine, 1902; Verhaar, 1950; Scales and Collins, 1972; Schachenmayr and Friede, 1978). They also found an increase in the number of fibres in the unaffected CST in these patients compared with the control subjects." etc.
